FOXP2 may be the gene that makes us human - or so the hype goes. Hyped or not, FOXP2 is rightly a focus of intense research, since it is a gene that clearly has a major effect on human speech. Mutations in FOXP2 are responsible for some rare but strange language disorders, such as the inability to learn grammatical skills or make the proper mouth/facial movements to properly articulate words. 

"Somebody's got to stand up to experts," cried the creationist head of the Texas State Board of Education, Don McLeroy. McLeroy's lament is nothing new in American culture - we love to lionize the artless hero who conquers the world through clean living and common sense, and without resorting to elitist expertise. (Although some see the pendulum swinging the other way.)

The key issue in considering competition is the question of whether changes in resource availability or mates would alter the confrontation. If the answer is negative, then no competition can be said to have occurred. The mere existence of confrontation does not necessarily denote competition.



So in considering competition the definition will be focused specifically on the conditions whereby two individuals engage each other in some confrontation for a specific and separate resource. To that end, one of the individuals will end up acquiring that resource while the other is either eliminated or goes elsewhere.



However, in a general sense, it is useful to consider first how competition actually manifests itself.

Many parasites depend on their host’s behavior in order to successfully reproduce. Instead of leaving this behavior to chance, some parasites actively manipulate their hosts to produce the desired behavior. For example, after infecting a rat, the taxoplasmosa gondii parasite needs to be transferred to a cat’s belly to reproduce. To do this, the sneaky parasite rewires its rat host to actively seek the smell of cat urine. When the rat gets eaten, the parasite completes its necessary transfer.

Cordyceps fungi infect insects and steer them to higher ground where, when the insect dies and the fungus bursts forth, the fungus spores will be more effectively dispersed by wind.

VIB (the Flanders Institute for Biotechnology) researchers linked to K.U.Leuven and Harvard University say they have shown that stretches of DNA previously believed to be useless 'junk' DNA play a vital role in the evolution of our genome. Their findings were that unstable pieces of junk DNA help tuning gene activity and enable organisms to quickly adapt to changes in their environments.

Junk DNA in two paragraphs

"Most people do not realize that all our genes only comprise about 3% of the total human genome. The rest is basically one large black box,"  says Kevin Verstrepen, heading the research team. "Why do we have this DNA, what is it doing?"

Oxidative stress has been linked to aging, cancer and other diseases in humans. Paradoxically, researchers have suggested that small exposure to oxidative conditions may actually offer protection from acute doses. Now, scientists at the University of California, San Diego, have discovered the gene responsible for this effect. Their study, published in PLoS Genetics on May 29, explains the underlying mechanism of the process that prevents cellular damage by reactive oxygen species (ROS).

Scientists at the University of Leeds say they have uncovered a previously unknown giant volcanic eruption.   And it's no ordinary eruption, they say - it led to global mass extinction 260 million years ago. 

The eruption in the Emeishan province of south-west China unleashed around half a million cubic kilometres of lava, covering an area 5 times the size of Wales, and wiping out marine life around the world.

Even if we don't always notice, our brain is constantly distracted with 'noise' - unimportant messages that are filtered out.   

When we pay attention, our neurons begin firing in harmony and a study in the May 29 issue of Science lays out what researchers say is the likely brain center that serves as the conductor of our neural chorus.  MIT neuroscientists say that neurons in the prefrontal cortex, the brain's planning center, fire in unison and send signals to the visual cortex to do the same, generating high-frequency waves that oscillate between these distant brain regions like a vibrating spring. These waves, also known as gamma oscillations, have long been associated with cognitive states like attention, learning, and consciousness.

Biomedical engineers at Boston University have taught bacteria how to count.  The researchers have wired a new sequence of genes that allow the microbes to count discrete events, opening the door for a host of potential applications, which could include drug delivery and sensing environmental hazards. 

The young but burgeoning field of synthetic biology addresses biological research questions with an engineering approach. Researchers design and build networks of genes, splicing them into bacterial genomes to run specific tasks or manufacture desired molecules – a process akin to installing biological computer software. Though the field is rapidly advancing, the gene-based tools available to synthetic biologists remain limited. 

How common is scientific misconduct?   It's a tough question to answer.   Scandals like Hwang Woo-Suk's faked stem-cell lines or Jon Sudbø's made-up cancer trials have demonstrated that fraudulent research is easy to publish, even in the most prestigious print journals, but are they deviations of a few "bad apples" or evidence that a great deal more is never discovered?  The actual numbers are a matter of dispute.

In a PLoS ONE meta-analysis of surveys questioning scientists about their misbehaviors, Daniele Fanelli of the University of Edinburgh, a PhD in the behavior and genetics of Malaysian and Panamanian tropical wasps, suggests that altering or making up data is more frequent than previously estimated and might be particularly high in medical research.