A team of scientists has identified a gene in rice that controls the size and weight of rice grains. The gene may prove to be useful for breeding high-yield rice and, thus, may benefit the vast number of people who rely on this staple food for survival.

The researchers first searched for and identified mutant strains of rice that exhibited underweight grains. "We found a particular mutant that is defective in its ability to produce normal-sized grains," said Zuhua He, a biology professor at the Chinese Academy of Sciences and the leader of the team. The group then examined the mutant and found that it carried a mutation within the GIF1 gene. "The GIF1 gene is responsible for controlling the activity of the enzyme invertase, which is located in the cell wall and converts sucrose to substances that then are used to create starch," said He. "Invertase is important in the formation of starch within developing grains of rice. If invertase is not active, the rice plant cannot produce edible grains."

Instead of using a flat microchip as the light sensor for their new camera, a team of engineers has developed a sensor that is a flexible mesh of wire-connected pixels. The mesh is made from many of the same materials as a standard digital-camera sensor, but has the unique ability to conform to convoluted, irregular surfaces.

The technology breakthrough is a novel approach that bypasses a traditional planar sensor of adjacent pixels and instead relies upon an array of pixels interconnected by small wires. Using a flexible, temporary backing, the researchers can form the array into a curved shape and then transfer the array to its permanent location affixed to a glass lens.

Over the last 20 years, many researchers have tried to manufacture such electronic eye systems, but until now, none were able to create a working camera.

A protein required for the earliest steps in embryonic development also plays a key role in solidifying fear memories in the brains of adult animals, scientists have revealed. An apparent "hub" for changes in the connections between brain cells, beta-catenin could be a potential target for drugs to enhance or interfere with memory formation.

The protein beta-catenin acts like a Velcro strap, fastening cells' internal skeletons to proteins on their external membranes that connect them with other cells. In species ranging from flies to frogs to mice, it also can transmit early signals that separate an embryo into front and back or top and bottom.

University of Utah engineers have devised a new way to slice thin wafers of germanium for use in efficient solar power cells. They say the new method should lower the cost of such cells by reducing the waste and breakage of the brittle semiconductor.

Germanium solar cells are expensive and so are only now used mainly on spacecraft but with the improved wafer-slicing method, "the idea is to make germanium-based, high-efficiency solar cells for uses where cost now is a factor," particularly for solar power on Earth, says Eberhard "Ebbe" Bamberg, an assistant professor of mechanical engineering. "You want to do it on your roof."

Dinesh Rakwal, a doctoral student in mechanical engineering, adds: "We're coming up with a more efficient way of making germanium wafers for solar cells – to reduce the cost and weight of these solar cells and make them defect-free."

Natural grass fires are evidently more important for the ecology of savannahs than has previously been assumed, according to the findings of a study carried out in Etosha National Park in the north of Namibia.

Writing in the Journal of Ecology, researchers from the Helmholtz Centre for Environmental Research (UFZ), the University of Frankfurt am Main and the University of Cologne say that fire plays an important role in regeneration.

It is the first study to have investigated the complex interplay of the factors fire, competition, moisture and seed availability in relation to a grass species. Periodic fires in semi-arid regions can lead to older tufts of grass disappearing, thereby making room for younger grasses. The findings are particularly significant for the management of semi-arid nature reserves, in which, in the absence of natural plant eaters, fires are the only practical means of renewing the grass canopy.

Researchers at UT Southwestern Medical Center have found that individuals who carry a specific form of the gene PNPLA3 have more fat in their livers and a greater risk of developing liver inflammation.

They also found that Hispanics are more likely to carry the gene variant responsible for higher liver-fat content than African-Americans and Caucasians.

The new findings, published in Nature Genetics, provide a gene-based explanation for the results of a 2004 UT Southwestern-led study that determined that the propensity to develop nonalcoholic fatty liver disease differs among ethnic groups, with a higher percentage of Hispanics developing the disorder than African-Americans or Caucasians.

In marking the passing of its founder, Paul Newman, the Association of Hole in the Wall Camps has issued the following statement:

While greatly saddened by Paul's passing, the family of Hole in the Wall Camps celebrates the life of Paul Newman and the legacy he has left us and the world.

Paul's commitment to the welfare of children suffering from serious illnesses and life-threatening diseases was never-ending. His infinite passion to provide kids who are sick with a place "to kick back and raise a little hell" has touched the lives of children everywhere.

Maybe its the fear of turning 40. Maybe its the feeling of unfinished business. Maybe its the fire in the belly that has not quite extinguished. For retired elite athletes, the itch is always there to make a return after experiencing "life after sport". For some, it becomes too strong to ignore.

This year has seen the return of at least three champions, Dara Torres, Lance Armstrong and Brett Favre. As they explain their individual reasons for coming back, some similarities emerge that have more to do with psychological needs than practical needs.

In a recent Miami Herald article, Torres explained her comeback to competitive swimming at age 41, "For me, it's not like I sat around and watched swimming on TV and thought, `Oh, I wish I was still competing'. It was more gradual. But all of a sudden, something goes off inside you and you start seriously thinking about a comeback. You'd think the competitive fire would die down with maturity, but I've actually gotten worse. I wasn't satisfied with silver medals. I hate to lose now more than I did in my 20s. I'm still trying to figure out why.''

David Sloan Wilson, over at the Huffington Post, has replied to my criticism of his previous essay on why the so-called “invisible hand” guiding financial markets is, as he puts it, “morally, ethically, spiritually, physically, positively, absolutely, undeniably and reliably dead.” David and I have a good working relationship (he is one of the infamous “Altenberg 16”) and mutual respect, so the following is meant to be in the spirit of an open minded debate between two scholars. David agrees, contrary to many of our colleagues, that it is good for the public to see scientists honestly argue their diverging positions in public, so here it goes.

First off, David claims that both conservative commentator Larry Arnhart and I “have objected to [David’s] declaration that the invisible hand is dead.” Arnhart has, I certainly have not. I completely agree that this pernicious idea has gone the way of the dodos in light of the events of recent years (at least from the Enron debacle on). I simply disagree that such a conclusion has anything whatsoever to do with hypotheses on the evolution of human morality and cognitive abilities.

Wilson challenges my claim that evolutionary psychology cannot tell us much, as a science, about the evolution of social human behavior. He rhetorically asks “Would [Massimo] make the same claim about astronomy, geology, and paleobiology? Past events leave traces in the present that can be pieced together to produce solid knowledge.”

We have many great anti-tumor drugs that can do a fantastic job destroying the molecular insides of tumor cells. There is, however, a major catch: tumors have a nasty habit of become drug resistant. Such is the case with the breast cancer chemotherapeutic agent docetaxel. This drug can be effective at stopping breast cancer, but unfortunately many tumors are docetaxel-resistant. 50% of breast cancer patients receiving their initial course of chemotherapy are resistant to docetaxel, and it gets worse for patients who have already had chemotherapy - 70-80% of patients who have already received chemotherapy don't respond to this drug.

Administering docetaxel to resistant patients obviously wastes time that could be spent on other treatments. It also causes needless suffering of side effects. But is there some way to predict in advance who is going to be resistant? Or better yet, is there something we can do to eliminate docetaxel resistance altogether?

A Japanese group from the Japanese National Cancer Research Institute set out to tackle this problem, and their encouraging results have been reported in Nature Medicine. These researchers discovered a gene that makes breast cancer cells resistant to docetaxel, and they used that knowledge to knock out the source of docetaxel resistance. Although this study was largely confined to petri dishes and mice, cancer researchers can now use this result to identify patients who won't respond to docetaxel, and they are ready to test this new therapy target in real human cancers.