A new study from Danish researchers has found that childless men have a lower risk of developing prostate cancer than fathers, and that, paradoxically, the more children a father has, the lower the risk of the disease. The study appears in the February 15, 2008 issue of CANCER.

Whether fatherhood can affect the risk of prostate cancer remains controversial. Evidence has suggested that childless men may be at lower risk of prostate cancer than men with children, and that men who father sons may be at lower risk than men with daughters only.

LAS VEGAS, January 7 /PRNewswire/ --

- BT Vision IPTV customers will soon be able to receive best in gaming, TV and movies, all through Xbox 360.

Oxytocin, a hormone involved in child-birth and breast-feeding, helps people recognize familiar faces, according to new research in the January 7 issue of The Journal of Neuroscience. Study participants who had one dose of an oxytocin nasal spray showed improved recognition memory for faces, but not for inanimate objects.

When we think of the genetic changes that had to take place during our evolutionary history, we typically think of changes that resulted in a gain of function, like genetic changes that resulted in a larger and more sophisticated brain, improved teeth for our changing prehistoric diet, better bone anatomy for bipedalism, better throat anatomy for speech, and so on. In many cases however, we have lost genes in our evolutionary history, and some of those losses have been beneficial. The most widely known example, found in every introductory biochemistry textbook, is the sickle-cell mutation in hemoglobin - a clear example of a mutation that damages a functional protein yet confers a beneficial effect. People with mutations in both copies of this particular gene are terribly sick, but those who have one good and one bad copy are more resistant to malaria. Another example is the CCR5 gene - people with mutations that damage this gene are more resistant to HIV. In the more distant past, a universal human mutation in a particular muscle gene that results in weaker jaw muscles may have played a role in brain evolution, by removing a constraint on skull dimensions. These few examples were found primarily by luck, but now with the availability of multiple mammalian genome sequences, researchers can systematically search for human genes that show signs of being adaptively lost at some point in our history. David Haussler's group at UC Santa Cruz, in a recent paper, looked for the genes we lost as we developed into our modern-day human species. What they found could help us better understand our evolutionary history, and possibly the human diseases that are the side-effects of that history.

Eight-year-old boys dream of being superheroes - flying high above the clouds with nothing to limit themselves but their imaginations - and Danner Cronise got his dream, courtesy of his dad, Ray, co-founder of Zero Gravity Corporation. In the process he set the record as the youngest person to experience zero-gravity flight.

Danner was joined by his father Ray, and older sisters Erin (10) and Alex (12). A NASA engineer for more than 15 years, Ray Cronise had a unique and personal reason for wanting to see his children enjoy weightlessness first hand. "It is awe inspiring to be able to take your kids on such a memorable experience. Being exposed to these kinds of adventures kindles curiosity and keeps them dreaming," said Ray.

A team of researchers at Penn Sate has used an animal model to reveal, for the first time, a physiological basis for the effect of alcohol on male sexual behavior, including increased sexual arousal and decreased sexual inhibition.

The research in PLoS ONE resulted in four novel findings with broad importance for further addiction research. It is the first study to characterize the effects of chronic alcohol exposure in fruit flies. "Physiological evidence supporting various theories about the effect of alcoholic drinks has been lacking, so our now having a suitable animal model makes it possible to conduct much-needed laboratory research on this issue," explains research-team-leader Kyung-An Han, associate professor of biology and a neuroscientist at Penn State.

A glitch in the mechanism by which cells recycle damaged components may trigger Parkinson’s disease, according to a study by scientists at the Albert Einstein College of Medicine of Yeshiva University. The research appears in The Journal of Clinical Investigation and could lead to new strategies for treating Parkinson’s and other neurodegenerative diseases.

All cells depend on a surveillance system known as autophagy (which literally means “self eating”) to digest and recycle the damaged molecules that arise as cells age. In autophagy, defective proteins and other molecules are transported to membrane-bound sacs called lysosomes. After attaching to the lysosomal membrane, the molecules enter the lysosome, where they are digested by enzymes.

A diagnosis of childhood cancer 50 years ago meant almost certain death. Now, because of scientific advances, the majority of newly diagnosed children can expect to survive.

Continued from Part 1: I interviewed Gary Taubes by phone a few weeks ago, shortly after he gave a talk about the main ideas of his new book — Good Calories, Bad Calories — at UC Berkeley. The interview lasted about 2 hours. This is part 2. SETH: What do you think about prions? GARY TAUBES: Here’s the problem with prions: the claim is that here’s a radical discovery — an infectious agent that doesn’t have nucleic acid — and it’s based fundamentally on a negative result, which is that when researchers have gone looking for the nucleic acids they failed to find them. Therefore, so the logic goes, they must not be there.

You may not think of oceanography when it comes to cancer research but an unexpected discovery in marine biomedical laboratories at the Scripps Institution of Oceanography has led to information on how a marine organism creates a natural product currently being tested to treat cancer in humans.